Filtros : "ICB" "ROSSONI, LUCIANA VENTURINI" "Experimental physiology" Removidos: "HU" "Nunes, Maria Tereza" "EE" Limpar

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  • Fonte: Experimental physiology. Unidades: ICB, EEFE

    Assuntos: FISIOLOGIA, SISTEMA CARDIOVASCULAR

    Acesso à fonteDOIComo citar
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    • ABNT

      COUTO, Gisele Kruger et al. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, v. 99, n. 10, p. 1427–1438, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2014.079236. Acesso em: 06 maio 2024.
    • APA

      Couto, G. K., Davel, A. P., Brum, P. C., & Rossoni, L. V. (2014). Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, 99( 10), 1427–1438. doi:10.1113/expphysiol.2014.079236
    • NLM

      Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 maio 06 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236
    • Vancouver

      Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 maio 06 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236

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